A woman tries holding an ice pack to her stomach to deal with ulcer pain.

Understanding the Staging of Pressure Ulcers

Stem Health Plus blogs.

Knowing the stages is more than a chart exercise—it’s how clinicians decide whether a reddened heel needs offloading and moisture care or whether a deep sacral wound requires debridement and infection control. The staging of pressure ulcers provides a shared language that guides priorities, communicates risk across disciplines, and sets realistic timelines for improvement. When teams classify accurately, they protect patients from under-treatment that lets tissue damage spread, and from over-treatment that can traumatize fragile skin. In practice, clear staging also supports documentation quality, aligns expectations with families, and anchors quality improvement efforts that reduce prevalence over time.

The Staging of Pressure Ulcers From Early Warning to Full-Thickness Injury

The framework moves from skin that looks intact but threatened, to partial-thickness loss, then to full-thickness loss that reaches into subcutaneous tissue and beyond. Stage 1 signals risk and calls for quick intervention; stage 2 shows shallow loss with exposed dermis; stage 3 extends through the dermis into the fat; stage 4 exposes structures such as tendons, muscles, or bones. Unstageable wounds hide their depth beneath slough or eschar, and a deep tissue pressure injury presents as maroon or purple discoloration that can evolve rapidly. Each category tells a different story about perfusion, pressure, and time under load, which is why interventions differ so much from stage to stage.

How Clinicians Examine Skin for the Staging of Pressure Ulcers

A careful look and light touch tell most of the tale. Assessors watch for changes in color, temperature, texture, and sensation, then evaluate depth, drainage, odor, and the condition of surrounding skin. The goal is to understand whether damage is superficial or full thickness, whether bioburden or moisture is complicating things, and where pressure and shear forces are acting so you can remove them. Consistency is key: reassessing under the same light, after relieving pressure, and with clean, dry skin reduces guesswork and improves agreement between observers on busy units.

Blanching Guides Early Calls


Pressing lightly and watching for color return helps distinguish reactive hyperemia from non-blanchable erythema.

Depth Determines the Stage

If subcutaneous tissue is involved, you’ve left the partial-thickness category and must classify higher.

Stage 1 Within the Staging of Pressure Ulcers

Stage 1 is characterized by intact skin with non-blanchable redness in a localized area, typically over a bony prominence. It can be warmer or cooler than the surrounding tissue, painful or tender, and may feel firmer or softer. This is where quick action pays off: offload pressure, manage microclimate, and address friction and shear. Because the skin barrier is intact, the focus is on prevention—repositioning, moisture management, and support surfaces—to stop progression before it becomes an open lesion that risks infection and scarring.

Stage 2 in the Staging of Pressure Ulcers

Stage 2 presents as a shallow open area with a pink to red wound bed and viable tissue; it might also appear as a serum-filled blister. What it is not: slough, eschar, or fat visible—those features push classification beyond partial thickness. At this level, moisture balance and gentle protection are the priorities. The aim is to preserve the surrounding skin, limit maceration, and provide an environment where epithelial cells can migrate cleanly across the surface without crusting or trauma at each dressing change.

Keep It Moist, Not Wet

Dressings should prevent desiccation while avoiding the sogginess that breaks down margins.

Friction Control Matters

Silicone interfaces and mindful transfers reduce shear that can deepen the defect.

Stage 3 Explained in the Staging of Pressure Ulcers

Stage 3 means the damage extends through the dermis into the subcutaneous tissue, often accompanied by undermining or tunneling. You will not see fascia, tendon, or bone, but you may see slough. Drainage can vary from scant to heavy, depending on the bioburden and location. Management pairs pressure redistribution with careful debridement of nonviable tissue when indicated, moisture balance to protect the wound bed and periwound skin, and vigilant infection surveillance. Nutrition and perfusion become crucial levers here because collagen synthesis and granulation require oxygen, protein, and micronutrients to rebuild volume safely.

Medical professional putting a bandage on a patient.

Stage 4 and the Staging of Pressure Ulcers

Stage 4 reveals fascia, tendon, muscle, cartilage, or bone, signaling deep destruction and prolonged pressure or shear. These wounds can hide pockets where fluid collects and bacteria flourish, making staging only the first step in a broader plan that addresses complexity. Offloading must be absolute, debridement methodical, and infection control proactive, often in collaboration with surgical or infectious disease teams. Healing horizons are longer, and goals may include preventing further deterioration when comorbidities limit the body’s ability to close large defects.

Know What You’re Seeing

Visible structure confirms the category regardless of wound size or patient discomfort.

Think Downstream Risk

Bone exposure raises concern for osteomyelitis and demands close clinical coordination.

Unstageable in the Staging of Pressure Ulcers

Sometimes a wound is covered by slough or eschar, which makes depth impossible to determine. Until the base is visible—by softening slough, lifting necrotic tissue where appropriate, or allowing stable heel eschar to act as a natural cover when it’s dry, adherent, and uninfected—you cannot accurately classify. The priorities are stabilizing the area, preventing contamination, and planning safe removal of barriers to assessment. Once the base is revealed, you may find a stage 3 or stage 4 lesion, and the plan shifts accordingly.

Deep Tissue Pressure Injury Within the Staging of Pressure Ulcers

A deep tissue presentation shows intact skin with persistent, non-blanchable, deep red, maroon, or purple discoloration, sometimes accompanied by a dark, blood-filled blister. It reflects damage in the underlying soft tissue caused by pressure or shear at the bone–muscle interface before the skin itself gives way. Because it can evolve quickly, early offloading is non-negotiable. Teams monitor closely, protect the area from friction, and address perfusion deficits so that a potentially hidden injury doesn’t develop into a larger, open wound.

Color Tells a Story

Hue changes paired with temperature or firmness shifts often precede surface breakdown.

Expect Evolution

Rapid change in size or depth after offloading means reassessment and plan updates.

Medical Devices and Moisture Injuries

Not every skin injury from care is a pressure category. Moisture-associated skin damage from incontinence or perspiration and adhesive-related injuries look different, follow different patterns, and need different fixes. Medical device–related pressure injuries often fit the framework, but they frequently occur in unusual shapes that mirror the device’s footprint. Recognizing these distinctions prevents misclassification and directs interventions to the actual cause—whether that’s improved moisture control, gentler removal techniques, or padding and repositioning of device components.

Risk Factors That Amplify the Staging of Pressure Ulcers Across Settings

Limited mobility, decreased sensation, poor perfusion, and malnutrition raise the probability that pressure and shear will outpace the body’s repair capacity. The staging system doesn’t list causes, but it makes risks visible because higher categories often cluster with hemodynamic instability, edema, and glycemic variability. It is rarely one factor; cumulative strain and time under load tip the balance. Addressing root issues—hydration, protein intake, offloading routines, and microclimate—gives the skin a chance to rebound before damage deepens.

Circulation Changes Outcomes

Edema control, warmth, and tobacco cessation improve oxygen delivery to threatened tissue.

Intake Fuels Repair

Adequate calories and protein support granulation and epithelial migration.

Documentation That Strengthens the Staging of Pressure Ulcers

Clear notes reduce disputes and delays. Location using anatomical landmarks, size in three dimensions, description of tissue types, presence of undermining or tunneling, exudate character, and the condition of the edges all help future readers visualize the wound accurately. Photographs—with consent and privacy safeguards—add consistency between shifts and support quality review. When documentation maps directly to the category criteria, care plans become easier to follow and modify, especially during transitions between units or facilities.

Treatment Decisions Linked to the Staging of Pressure Ulcers

Because stages reflect depth and tissue involvement, they hint at which levers will move healing. Early categories emphasize aggressive offloading, moisture balance, and friction reduction so superficial damage can regress. Full-thickness categories add debridement strategy, infection control, and attention to biofilm that stalls granulation. Support surfaces scale with risk, from high-specification foam to alternating pressure or low-air-loss beds when indicated. Pair these with nutrition, pain management, and patient education to make gains stick between visits.

Offload First

No dressing compensates for continuous pressure on a bony prominence.

Keep Edges Healthy

Protect periwound skin to prevent maceration, which can enlarge the wound footprint.

Pitfalls That Muddy the Staging of Pressure Ulcers and Slow Progress

Common missteps include calling a moist intertriginous rash a pressure lesion, assuming every dark heel is full-thickness damage, or staging a wound before removing loose slough that hides the base. Others involve mistaking adhesive trauma for friction injury or ignoring shear from sliding down in bed. These errors matter because they lead to mismatched treatments and missed opportunities to relieve the actual stressor. Slowing down to reassess after offloading and gentle cleansing often corrects the picture and realigns the plan.

Education, Teamwork, and the Future of the Staging of Pressure Ulcers

Staging stays useful only if teams continue to learn together. Short huddles to calibrate assessments, bedside teaching moments on blanching and depth cues, and shared photo libraries with de-identified examples all raise reliability. As support surfaces, dressings, and diagnostic tools evolve, the framework will keep serving as the map that links assessment to action. The aim is simple: fewer new lesions, faster recovery when they occur, and patients who feel the difference because everyone is speaking the same language and pulling in the same direction.
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